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Results Selected demographic, anthropometric and some obstetric characteristics of the two study groups are shown in Table I. The mean age of patients at implant insertion was 27.9 0.7 and 27.1 0.6 years for Implanon and Jadelle users, respectively. There were no significant differences in the pre-insertion characteristics of women in the two groups. In both groups, almost 40% of the women were housewives, 45% worked in retail or services, while 15% were professional workers. Seventy-seven percent of Implanon users and 82% of Jadelle users were white. Almost an equal proportion of women had used hormonal or non-hormonal contraceptive methods as their last method before the insertion of the implant in both groups, including DMPA used by four and five women in the Implanon and Jadelle groups, respectively. Fourteen women in the.
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Kim, I. H., MD . 158, 255 Kim, James J., MD . 102, 110 Kim, Jay J., MD . 144, 231 Kimmey, Gerrit A., MD . 95 Kincaid, Christine, MD . 174 Kinder, Jack L., MD . 128 Kinetic Healthcare . 27, 62 King Watts, Susan C., CNM . 16 King, Devin A., MD . 205 King, Donald G., OD . 116 King, John H., MD . 111 King, Karla R., PT . 12 King, R. Keith, OD . 116 King, Richard W., MD . 112 King, Roger E., MD . 190 King, Tamela R., CRNP . 15 King, Teann Marie, LPC . 286 Kinkade, Lincoln N., PT. 42 Kinney, John H., MD . 254 Kinsey, Gail, LPC. 284 Kirchdoerfer, Elaine J., MD . 219 Kircher, Christopher, MD. 255 Kirk, Hanno W., LICSW . 285 Kirk, Michael D., MD . 219 Kirkhart, Laurel A., MD . 255 Kirlangitis, John I., PT . 22 Kiser Crouch, Peggy L., DC . 121 Kish, Shari L., SLP . 71 Kisner, Amy L., MD. 128 Kisner, Michael L., DC . 76 Kissel, Joy H., OD . 231 Kissel, Kevin T., OD . 231 Kissinger, Mark A., DO . 257 Kitchin, Llewellyn I., MD . 235 Kitiphongspattana, Kriengkrai, MD . 150 Kitts, Ellen L., MD . 199, 249 Kitzmiller, Melissa D., MD . 110 Kiziminski, Kelly J., SLP . 50, 69 Klay, John W., MD. 201 Klein, Carol A., MD . 94, 282 Klein, Jeremy C., MD. 275 Klein, Robert J., PhD . 294 Klennert, Benjamin J., PA-C . 52.
PSM02-1 Vatassery , G.T. Modulation of vitamin E alpha tocopherol ; in brain by apolipoprotein E PSM02-2 Lajtha, A. Neurotransmitter Changes by Drugs Used in Alzheimer Therapy PSM02-3 Walton, H.S. Real-time quantitative RT-PCR analysis of EAAT2 splice variants PSM02-4 Gibson, G.E. Select antioxidants modify endoplasmic reticulum calcium stores, capacitative calcium entry and mitochondrial calcium export PSM02-5 Chen, C. Visualization of APP dimerization and APP Notch heterodimerization in living cells using Bimolecular Fluorescence Complementation PSM02-6 Chauhan, N.B. Cholinergic immunolesioning produced tangle-like inclusions in TgCRND8 Brain PSM02-7 Vasilevko, V. Affects of peripheral pro- and anti-inflammatory agents on glial activation and clearance of amyloid in transgenic models of AD PSM02-8 Petrushina, I. Mannan-Ab28 induced an anti-inflammatory anti-Ab immune response in APP Tg mice and increased cerebrovascular microhemorrhages PSM02-9 Copenhaver, P.F. The insect homologue of the Amyloid Precursor Protein may regulate neuronal migration in the developing nervous system.
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Ments reduce the risk of stroke recurrence. Hypertensive patients 50 years of age who suffered from ischemic stroke 1 month before the visit to participating hospitals are to be included. Those who have serious cardiac, renal, or hepatic diseases, aneurysms, or malignancies are excluded. Patients are randomized into the following three groups: 1 ; those treated with a calcium antagonist eg, nilvadipine ; , 2 ; those treated with an angiotensin-converting enzyme inhibitor eg, cilazapril ; , and 3 ; untreated patients. The primary end points are stroke, other cardiovascular diseases, sudden death, and renal failure. A total of 800 patients will be recruited and followed for at least 3 years. Principal Investigators: M. Fujishima, MD Contact: M. Fujishima, MD, Second Department of Internal Medicine, Faculty of Medicine, Kyushu University, Maidashi 3-1-1, Fukuoka 812-8582, Japan. Phone 81-92-642-5256. Fax 81-92-642-5271. Location: The SHARK Study Center Number of Centers: 54 recruitment will continue until December 31, 1999 ; Dates of Study: September 1995 through December 2002.
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P17-4 MOLECULAR MODELING, SYNTHESIS, AND CHARACTERIZATION OF RADIOIODINATED QUINAZOLINONE DERIVATIVES FOR ENZYME-MEDIATED BREAST CANCER DIAGNOSIS AND THERAPY A. I. Kassis, K. Chen, K. Wang, A. F. Al Aowad, S. J. Adelstein Department of Radiology, Harvard Medical School, Boston, MA GENETICALLY TARGETED RADIOTHERAPY USING SODIUM IODIDE SYMPORTER IN BREAST CANCER CELLS K. Krager, A. Gaut, M. Madsen, D. Trask, M. Graham, F. Domann Departments of Radiology, Radiation Oncology, and Otolaryngology, University of Iowa Hospitals and Clinics, Iowa City, IA SEGMENT-BASED BREAST IMRT USING A GENETIC DOSE OPTIMIZATION ALGORITHM T. Li, C. Cotrutz, D. Gofinett, L. Xing Department of Radiation Oncology, Stanford University, Stanford, CA CHARACTERIZATION OF HELICAL ELECTRON BEAMS FOR BREAST RADIATION THERAPY L. Ma University of Maryland School of Medicine, Baltimore, MD TREATMENT RELATED CARDIAC TOXICITY IN PATIENTS TREATED FOR BREAST CANCER L. B. Marks Duke University Medical Center, Durham, NC ACTIVATION OF ESTROGEN RECEPTOR-ALPHA BY DNA METHYLTRANSFERASE AND HISTONE DEACETYLASE INHIBITORS RADIOSENSITIZES HUMAN ESTROGEN RECEPTORALPHA NEGATIVE BREAST CANCER CELLS A. Munshi, T. A. Bucchholz, R. E. Meyn Department of Experimental Radiation Oncology, University of Texas M.D. Anderson Cancer Center, Houston, TX P17-10 DEVELOPING BIOMARKERS OF RADIATION RESPONSE: THE CORRELATION BETWEEN PLASMA TGF-BETA AND FRACTION SIZE A. Bevan, C. Minami, C. Park University of California at San Francisco, San Francisco, CA P17-11 RESISTANCE TO IRRADIATION CYTOTOXICITY AND INCREASED TRANSFORMATION IN BREAST DUCTAL CARCINOMAS REQUIRES PROTEIN SYNTHESIS AND IS MEDIATED BY ELEVATED LEVELS OF THE INITIATION FACTOR EIF4G S. Braunstein, 1 S. Formenta, 2 K. Karpisheva, 1 R. J. Schneider1 Departments of 1Microbiology and 2Radiation Oncology, New York University School of Medicine, New York, NY P18-2.
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Samenvatting Er bestaan verschillende meningen over de manier waarop farmacochemie in de praktijk uitgevoerd zou moeten worden. De in dit proefschrift beschreven procedure voor het ontwerpen van nieuwe geneesmiddelen bestaat uit verschillende fases, t.w. de selectie van een template verbinding, de experimentele proefopzet, de synthese van nieuwe verbindingen, de in vitro farmacologische evaluatie, het molecular modelen en de multivariate statistische evaluatie. Op elk van deze fases is reeds in meerdere of mindere mate de aandacht gevestigd geweest, maar in dit proefschrift wordt de meeste nadruk gelegd op de multivariate statistische evaluatie. In de zoektocht naar potente en selectieve D3 antagonisten werd in eerste instantie de trans-N n-Propyl ; -7-[[trifluoromethyl ; sulfonyl]oxy]-OHB[f]Q zie 1 in Tabel 3.3 ; verondersteld een template verbinding te zijn voor dit onderzoek. De verbinding vertoonde presynaptisch DA receptor antagonistische eigenschappen in ratten zie Tabel 1.4 ; , ondanks het feit dat de 7-hydroxy-analoog een potente agonist is. Het racemaat 1 vertoonde een tienvoudige selectiviteit voor de DA D3 receptor boven de D2 receptor. De invloed van de 7-triflaatgroep op deze effecten was van speciaal belang. Verschillende OHB[f]Qs werden ontworpen en beschreven met theoretische fysisch-chemische variabelen, terwijl verbinding 1 werd gebruikt als template. Slechts een fractie van de meest verschillende verbindingen werd geselecteerd door middel van een experimentele proefopzet in the descriptor space zie Hoofdstuk 3 ; . Derhalve werden de 16 verbindingen gesynthetiseerd en getest op in vitro affiniteit voor de DA D2, D3 en D4 receptor subtypes. Geen van de verbindingen was echt selectief voor n van deze receptoren; in het algemeen vertoonden verbindingen met een hydroxygroep op de 7-positie een significante hoge affiniteit voor alle drie dopamine receptoren, terwijl de verbindingen met de sulfon ester groep minder potent waren. Bovendien onderdrukte de sulfon ester groep de affiniteit voor de DA D4 receptor. De stikstof substituent mag zo groot zijn als een fenylethyl groep zonder dat de affiniteit voor de DA receptors nadelig wordt benvloed. Uiteindelijk ontbreekt bij een verbinding met een 7-OH groep en een N-propargyl groep de affiniteit voor de DA D4 receptor. De enigszins starre N-propargyl groep en de lage pKa-waarde 6.1 ; zouden bijdragende factoren kunnen zijn voor de lage D4 affiniteit. Op dit punt zou een 3D QSAR model informatie kunnen verschaffen over de verder te volgen procedure; welke volgende verbinding dient te worden gesynthetiseerd? Echter, door gebrek aan tijd werd verder onderzoek naar deze data set niet voortgezet. In plaats daarvan werd een data set ter beschikking gesteld door Dr. Shelly Glase Parke-Davis, VS ; waarmee de multivariate statistische analyses werden onderzocht. Dit theoretische deel van het proefschrift is hoofdzakelijk gericht op de optimalisering van multivariate en multiweg regressie-analyse methoden in 3D QSAR. Hoofdstuk 4 beschrijft hoe de conformatie-analyses en de alignments van wederzijdse en mogelijke interactie-punten met de DA D3 receptor van de flexibele verbindingen van Dr. Glase werden uitgevoerd. De absolute configuratie van de verbinding 1 in Tabel 4.1 ; , die werd gebruikt als template voor de resterende 29 verbindingen, werd bepaald met behulp van rntgen 155 and dulcolax.
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787. Oral H, Scharf C, Chugh A, et al. Catheter ablation for paroxysmal atrial fibrillation: segmental pulmonary vein ostial ablation versus left atrial ablation. Circulation 2003; 108: 235560. Cappato R, Calkins H, Chen SA, et al. Worldwide survey on the methods, efficacy, and safety of catheter ablation for human atrial fibrillation. Circulation 2005; 111: 11005. Nademanee K, McKenzie J, Kosar E, et al. A new approach for catheter ablation of atrial fibrillation: mapping of the electrophysiologic substrate. J Coll Cardiol 2004; 43: 204453. Hsu LF, Jais P, Sanders P, et al. Catheter ablation for atrial fibrillation in congestive heart failure. N Engl J Med 2004; 351: 237383. Pappone C, Rosanio S, Augello G, et al. Mortality, morbidity, and quality of life after circumferential pulmonary vein ablation for atrial fibrillation: outcomes from a controlled nonrandomized longterm study. J Coll Cardiol 2003; 42: 18597. Marshall HJ, Harris ZI, Griffith MJ, et al. Prospective randomized study of ablation and pacing versus medical therapy for paroxysmal atrial fibrillation: effects of pacing mode and mode-switch algorithm. Circulation 1999; 99: 158792. Natale A, Zimerman L, Tomassoni G, et al. AV node ablation and pacemaker implantation after withdrawal of effective rate-control medications for chronic atrial fibrillation: effect on quality of life and exercise performance. Pacing Clin Electrophysiol 1999; 22: 16349. Marshall HJ, Harris ZI, Griffith MJ, et al. Atrioventricular nodal ablation and implantation of mode switching dual chamber pacemakers: effective treatment for drug refractory paroxysmal atrial fibrillation. Heart 1998; 79: 5437. Bubien RS, Knotts-Dolson SM, Plumb VJ, et al. Effect of radiofrequency catheter ablation on health-related quality of life and activities of daily living in patients with recurrent arrhythmias. Circulation 1996; 94: 158591. Anselme F, Saoudi N, Poty H, et al. Radiofrequency catheter ablation of common atrial flutter: significance of palpitations and quality-of-life evaluation in patients with proven isthmus block. Circulation 1999; 99: 53440. Lee SH, Tai CT, Yu WC, et al. Effects of radiofrequency catheter ablation on quality of life in patients with atrial flutter. J Cardiol 1999; 84: 27883. Hindricks G, Piorkowski C, Tanner H, et al. Perception of atrial fibrillation before and after radiofrequency catheter ablation: relevance of asymptomatic arrhythmia recurrence. Circulation 2005; 112: 30713. Senatore G, Stabile G, Bertaglia E, et al. Role of transtelephonic electrocardiographic monitoring in detecting short-term arrhythmia recurrences after radiofrequency ablation in patients with atrial fibrillation. J Coll Cardiol 2005; 45: 8736. Karch MR, Zrenner B, Deisenhofer I, et al. Freedom from atrial tachyarrhythmias after catheter ablation of atrial fibrillation: a randomized comparison between 2 current ablation strategies. Circulation 2005; 111: 287580. Haissaguerre M, Jais P, Shah DC, et al. Electrophysiological end point for catheter ablation of atrial fibrillation initiated from multiple pulmonary venous foci. Circulation 2000; 101: 140917. Ren JF, Marchlinski FE, Callans DJ, et al. Increased intensity of anticoagulation may reduce risk of thrombus during atrial fibrillation ablation procedures in patients with spontaneous echo contrast. J Cardiovasc Electrophysiol 2005; 16: 4747. Pappone C, Oral H, Santinelli V, et al. Atrio-esophageal fistula as a complication of percutaneous transcatheter ablation of atrial fibrillation. Circulation 2004; 109: 27246. Scanavacca MI, D'avila A, Parga J, et al. Left atrial-esophageal fistula following radiofrequency catheter ablation of atrial fibrillation. J Cardiovasc Electrophysiol 2004; 15: 9602. Mesas CE, Pappone C, Lang CC, et al. Left atrial tachycardia after circumferential pulmonary vein ablation for atrial fibrillation: electroanatomic characterization and treatment. J Coll Cardiol 2004; 44: 10719. Pappone C, Manguso F, Vicedomini G, et al. Prevention of iatrogenic atrial tachycardia after ablation of atrial fibrillation: a prospective randomized study comparing circumferential pulmonary vein ablation with a modified approach. Circulation 2004; 110: 303642. Andersen HR, Nielsen JC, Thomsen PE, et al. Long-term follow-up of patients from a randomised trial of atrial versus ventricular pacing for sick-sinus syndrome. Lancet 1997; 350: 12106. Connolly SJ, Kerr CR, Gent M, et al. Effects of physiologic pacing versus ventricular pacing on the risk of stroke and death due to cardiovascular.
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As a condition of membership, all schools shall adopt policies prohibiting the use and abuse of androgenic anabolic steroids. All member schools shall have participating students and their parents caregiver agree that the athlete will not use steroids without the written prescription of a fully licensed physician as recognized by the AMA ; to treat a medical condition. NOTE: Article 1 12 ; A Revised May 2005 Federated Council and duragesic
Tyrosine phosphorylation of STAT-1, -3, and -5 proteins, following leptin 5 nm ; treatment, is shown in Fig. 7. The concentrations of leptin used in this experiment were equivalent to those observed in obese subjects 40 ; . Tyrosine phosphorylation of STAT-3 and -5 was markedly elevated at 5 min, whereas phosphorylation of STAT-1 was not observed under the same experimental conditions.
13. Gupta, D., Kirkland, T. N., Viriyakosol, S., Dziarski, R. 1996 ; CD14 is a cell-activating receptor for bacterial peptidoglycan. J. Biol. Chem. 271, 23310 23316. Li, X., Bradford, B. U., Dalldorf, F., Goyert, S. M., Stimpson, S. A., Thurman, R. G., Makarov, S. S. 2004 ; CD14 mediates the innate immune responses to arthritopathogenic peptidoglycan-polysaccharide complexes of Gram-positive bacterial cell walls. Arthritis Res. Ther. 6, R273R281. 15. Vignal, C., Guerardel, Y., Kremer, L., Masson, M., Legrand, D., Mazurier, J., Elass, E. 2003 ; Lipomannans, but not lipoarabinomannans, purified from Mycobacterium chelonae and Mycobacterium kansasii induce TNFand IL-8 secretion by a CD14-Toll-like receptor 2-dependent mechanism. J. Immunol. 171, 2014 2023. Devitt, A., Moffatt, O. D., Raykundalia, C., Capra, J. D., Simmons, D. L., Gregory, C. D. 1998 ; Human CD14 mediates recognition and phagocytosis of apoptotic cells. Nature 392, 505509. 17. Grunwald, U., Fan, X., Jack, R. S., Workalemahu, G., Kallies, A., Stelter, F., Schutt, C. 1996 ; Monocytes can phagocytose Gram-negative bacteria by a CD14-dependent mechanism. J. Immunol. 157, 4119 4125. Casten, L. A., Pierce, S. K. 1988 ; Receptor-mediated B cell antigen processing. Increased antigenicity of a globular protein covalently coupled to antibodies specific for B cell surface structures. J. Immunol. 140, 404 410. Carayanniotis, G., Barber, B. H. 1987 ; Adjuvant-free IgG responses induced with antigen coupled to antibodies against class II MHC. Nature 327, 59 61. Zanetti, M. 1992 ; Antigenized antibodies. Nature 355, 476 477. Bona, C., Brumeanu, T. D., Zaghouani, H. 1994 ; Immunogenicity of microbial peptides grafted in self immunoglobulin molecules. Cell. Mol. Biol. Noisy-le-grand ; 40 Suppl. 1 ; , 2130. 22. Lunde, E., Munthe, L. A., Vabo, A., Sandlie, I., Bogen, B. 1999 ; Antibodies engineered with IgD specificity efficiently deliver integrated T-cell epitopes for antigen presentation by B cells. Nat. Biotechnol. 17, 670 675. Lunde, E., Western, K. H., Rasmussen, I. B., Sandlie, I., Bogen, B. 2002 ; Efficient delivery of T cell epitopes to APC by use of MHC class II-specific Troybodies. J. Immunol. 168, 2154 2162. Eidem, J. K., Rasmussen, I. B., Lunde, E., Gregers, T. F., Rees, A. R., Bogen, B., Sandlie, I. 2000 ; Recombinant antibodies as carrier proteins for sub-unit vaccines: influence of mode of fusion on protein production and T-cell activation. J. Immunol. Methods 245, 119 131. Rasmussen, I. B., Lunde, E., Michaelsen, T. E., Bogen, B., Sandlie, I. 2001 ; The principle of delivery of T cell epitopes to antigen-presenting cells applied to peptides from influenza virus, ovalbumin, and hen egg lysozyme: implications for peptide vaccination. Proc. Natl. Acad. Sci. USA 98, 10296 10301. Rtnes, J. S., Bogen, B. 1994 ; Ca2 mobilization in physiologically stimulated single T cells gradually increases with peptide concentration analog signaling ; . Eur. J. Immunol. 24, 851 858. Lunde, E., Bogen, B., Sandlie, I. 1997 ; Immunoglobulin as a vehicle for foreign antigenic peptides immunogenic to T cells. Mol. Immunol. 34, 11671176. 28. Norderhaug, L., Olafsen, T., Michaelsen, T. E., Sandlie, I. 1997 ; Versatile vectors for transient and stable expression of recombinant antibody molecules in mammalian cells. J. Immunol. Methods 204, 77 87. Neuberger, M. S. 1983 ; Expression and regulation of immunoglobulin heavy chain gene transfected into lymphoid cells. EMBO J. 2, 1373 1378. Van Voorhis, W. C., Steinman, R. M., Hair, L. S., Luban, J., Witmer, M. D., Koide, S., Cohn, Z. A. 1983 ; Specific antimononuclear phagocyte monoclonal antibodies. Application to the purification of dendritic cells and the tissue localization of macrophages. J. Exp. Med. 158, 126 145. Sallusto, F., Lanzavecchia, A. 1994 ; Efficient presentation of soluble antigen by cultured human dendritic cells is maintained by granulocyte macrophage colony-stimulating factor plus interleukin 4 and downregulated by tumor necrosis factor . J. Exp. Med. 179, 1109 1118. Shimazu, R., Akashi, S., Ogata, H., Nagai, Y., Fukudome, K., Miyake, K., Kimoto, M. 1999 ; MD-2, a molecule that confers lipopolysaccharide responsiveness on Toll-like receptor 4. J. Exp. Med. 189, 17771782. 33. Lanzavecchia, A. 1985 ; Antigen-specific interaction between T and B cells. Nature 314, 537539. 34. St Pierre, Y., Watts, T. H. 1990 ; MHC class II-restricted presentation of native protein antigen by B cells is inhibitable by cycloheximide and brefeldin A. J. Immunol. 145, 812 818 and echinacea.
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Illustrating these multiple sounds. The auscultatory clues suggesting ventricular tachycardia previously emphasized are a changing intensity of the first heart sound, slightly irregular ventricular rate, and lack of slowing of the ventricular rate with carotid sinus stimulation. In addition to these important findings, which can be observed on auscultation at the bedside, the hearing of multiple low-frequency sounds can aid in even more rapid suspicion of the diagnosis of ventricular tachycardia. Figure 3 shows 13 examples of tachycardia other than ventricular tachycardia, in which these multiple sounds are not present. As to the mechanism of these multiple sounds, further light can be shed on the problem by the observation of splitting of heart sounds with right or left bundle-branch block in the presence of congestive failure. In bundle-branch block the second heart sound is generally widely split, and frequently the first heart sound also. If the patient has congestive failure a ventricular diastolic gallop is usually present, and if first-degree heart block or P-R interval on the longer side ; or hypertension is present, atrial sounds may be heard. Figure 4 represents a patient with left bundle-branch block and cardiac decompensation, illustrating the wide splitting of the first and second heart sounds plus both atrial and ventricular diastolic gallops and dronabinol
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